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All considered, these data strongly suggest that SAMe depletion contributes to multiple pathophysiological processes observed in the AD brain, including the unsilencing of genes involved in AD development and progression, the depletion of acetylcholine, and the inability to appropriately dephosphorylate tau

Wu X, Hammer JA

*Correspondence: Nady Braidy [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Oleocanthal, a phenolic derived from virgin olive oil: a review of the beneficial effects on inflammatory disease